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Abstract Background Discrepancies in the utilization of reactive oxygen species (ROS) between cancer cells and their normal counterparts constitute a pivotal juncture for the precise treatment of cancer, delineating a noteworthy trajectory in the field of targeted therapies

Many anticancer agents induce apoptosis via activation of the MAP kinase pathway, specifically via JNK and p38 (Dav is, 2000

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Sixth degree burns include destruction of the epidermis, dermis, subcutaneous tissue and muscle